A RetroSearch Logo

Home - News ( United States | United Kingdom | Italy | Germany ) - Football scores

Search Query:

Showing content from https://link.springer.com/doi/10.1007/s11920-006-0042-y below:

The genetics of ADHD: A literature review of 2005

  • Faraone SV, Sergeant J, Gillberg C, Biederman J: The worldwide prevalence of ADHD: Is it an American condition? World Psychiatry 2003, 2:104–113.

    PubMed  Google Scholar 

  • Faraone SV, Perlis RH, Doyle AE, et al.: Molecular genetics of attention deficit hyperactivity disorder. Biol Psychiatry 2005, 57:1313–1323.

    Article  PubMed  CAS  Google Scholar 

  • Faraone S, Khan S: Candidate gene studies of attention deficit activity disorder, J Clin Psychiatry 2006, In press.

  • Bakker SC, van der Meulen EM, Buitelaar JK, et al.: A whole-genome scan in 164 Dutch sib pairs with attention-deficit/hyperactivity disorder: suggestive evidence for linkage on chromosomes 7p and 15q. Am J Hum Genet 2003, 72:1251–1260.

    Article  PubMed  CAS  Google Scholar 

  • Smalley SL, Kustanovich V, Minassian SL, et al.: Genetic linkage of attention-deficit/hyperactivity disorder on chromosome 16p13, in a region implicated in autism. Am J Hum Genet 2002, 71:959–963.

    Article  PubMed  Google Scholar 

  • Fisher SE, Francks C, McCracken JT, et al.: A genomewide scan for loci involved in attention-deficit/hyperactivity disorder. Am J Hum Genet 2002, 70:1183–1196.

    Article  PubMed  CAS  Google Scholar 

  • Arcos-Burgos M, Castellanos FX, Pineda D, et al.: Attention-deficit/hyperactivity disorder in a population isolate: linkage to loci at 4q13.2, 5q33.3, 11q22, and 17p11. Am J Hum Genet 2004, 75:998–1014.

    Article  PubMed  CAS  Google Scholar 

  • Arnsten AF, Li BM: Neurobiology of executive functions: catecholamine influences on prefrontal cortical functions. Biol Psychiatry 2005, 57:1377–1384. This article discusses neurobiological theories of prefrontal deficits thought to be associated with executive function. It goes into much detail regarding this brain region thought to be implicated in ADHD symptoms.

    Article  PubMed  CAS  Google Scholar 

  • Bobb AJ, Addington AM, Sidransky E, et al.: Support for association between ADHD and two candidate genes: NET1 and DRD1. Am J Med Genet B Neuropsychiatr Genet 2005, 134:67–72.

    PubMed  Google Scholar 

  • Hamarman S, Fossella J, Ulger C, et al.: Dopamine receptor 4 (DRD4) 7-repeat allele predicts methylphenidate dose response in children with attention deficit hyperactivity disorder: a pharmacogenetic study. J Child Adolesc Psychopharmacol 2004, 14:564–574. This is one of the few studies done that measured response criteria as they related to a particular allele. This study prospectively evaluated the possible relation between a treatment and a genotype.

    Article  PubMed  Google Scholar 

  • Kirley A, Lowe N, Mullins C, et al.: Phenotype studies of the DRD4 gene polymorphisms in ADHD: association with oppositional defiant disorder and positive family history. Am J Med Genet B Neuropsychiatr Genet 2004, 131:38–42.

    Article  PubMed  Google Scholar 

  • Lowe N, Kirley A, Mullins C, et al.: Multiple marker analysis at the promoter region of the DRD4 gene and ADHD: evidence of linkage and association with the SNP -616. Am J Med Genet B Neuropsychiatr Genet 2004, 131:33–37.

    Article  PubMed  Google Scholar 

  • Leung PW, Lee CC, Hung SF, et al.: Dopamine receptor D4 (DRD4) gene in Han Chinese children with attention-de.-cit/hyperactivity disorder (ADHD): increased prevalence of the 2-repeat allele, Am J Med Genet B Neuropsychiatr Genet 2005, 133:54–56. This study discusses ethnic variation in ADHD. Although the phenotypic symptoms of ADHD may be similar, Chinese Han subjects reported in this article had an increased prevalence of a less common allele of DRD4.

    PubMed  Google Scholar 

  • Stein MA, Waldman ID, Sarampote CS, et al.: Dopamine transporter genotype and methylphenidate dose response in children with ADHD. Neuropsychopharmacology 2005, 30:1374–1382. This is one of the few studies that examined the 9-repeat allele of DAT and suggested a relationship with pharmacologic response. In its prospective design, the ADHD subjects with a 9-repeat DAT allele were shown to have blunted MPH response compared with those subjects without the allele.

    Article  PubMed  CAS  Google Scholar 

  • Winsberg BG, Comings DE: Association of the dopamine transporter gene (DAT1) with poor methylphenidate response. J Am Acad Child Adolesc Psychiatry 1999, 38:1474–1477.

    Article  PubMed  CAS  Google Scholar 

  • Cornish KM, Manly T, Savage R, et al.: Association of the dopamine transporter (DAT1) 10/10-repeat genotype with ADHD symptoms and response inhibition in a general population sample. Mol Psychiatry 2005, 10:686–698.

    Article  PubMed  CAS  Google Scholar 

  • Hebebrand J, Dempfle A, Saar K, et al.: A genome-wide scan for attention-deficit/hyperactivity disorder in 155 German sib-pairs. Mol Psychiatry 2006, 11:196–205.

    Article  PubMed  CAS  Google Scholar 

  • Curran S, Purcell S, Craig I, et al.: The serotonin transporter gene as a QTL for ADHD, Am J Med Genet B Neuropsychiatr Genet 2005, 134:42–47.

    PubMed  Google Scholar 

  • Li J, Wang Y, Zhou R, et al.: Serotonin 5-HT1B receptor gene and attention deficit hyperactivity disorder in Chinese Han subjects. Am J Med Genet B Neuropsychiatr Genet 2004, 132:59–63.

    Google Scholar 

  • Domschke K, Sheehan K, Lowe N, et al.: Association analysis of the monoamine oxidase A and B genes with attention deficit hyperactivity disorder (ADHD) in an Irish sample: preferential transmission of the MAO-A 941G allele to affected children. Am J Med Genet B Neuropsychiatr Genet 2005, 134:110–114.

    PubMed  Google Scholar 

  • Loo SK, Specter E, Smolen A, et al.: Functional effects of the DAT1 polymorphism on EEG measures in ADHD. J Am Acad Child Adolesc Psychiatry 2003, 42:986–993. This study examined the neuropsychological measure of EEG and correlated it with the presence of a particular DAT polymorphism. This is important, as it touches upon a possible relationship between EEG differences in subjects possessing this polymorphism.

    Article  PubMed  Google Scholar 

  • Doyle AE, Willcutt EG, Seidman LJ, et al.: Attention-deficit/ hyperactivity disorder endophenotypes. Biol Psychiatry 2005, 57:1324–1335. This article provides a very thorough overview and examples of endophenotypes and discusses their relevance in studies of ADHD. It also discusses the relevance of understanding endophenotypes as a component of the ADHD disease state.

    Article  PubMed  CAS  Google Scholar 

  • Owens EB, Hinshaw SP, Kraemer HC, et al.: Which treatment for whom for ADHD? Moderators of treatment response in the MTA. J Consult Clin Psychol 2003, 71:540–552.

    Article  PubMed  Google Scholar 

  • Roman T, Schmitz M, Polanczyk GV, et al.: Is the alpha-2A adrenergic receptor gene (ADRA2A) associated with attention-deficit/hyperactivity disorder? Am J Med Genet B Neuropsychiatr Genet 2003, 120:116–120.

    Article  PubMed  Google Scholar 

  • Volk HE, Neuman RJ, Todd RD: A systematic evaluation of ADHD and comorbid psychopathology in a population-based twin sample. J Am Acad Child Adolesc Psychiatry 2005, 44:768–775.

    Article  PubMed  Google Scholar 


  • RetroSearch is an open source project built by @garambo | Open a GitHub Issue

    Search and Browse the WWW like it's 1997 | Search results from DuckDuckGo

    HTML: 3.2 | Encoding: UTF-8 | Version: 0.7.4