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Brief review of available evidence concerning the potential induction of genomic damage by methylphenidate

Summary.

Children affected by attention deficit/hyperactivity disorder are often treated with methylphenidate (MPH). Two years ago, an increase in genomic damage after 3 months of MPH treatment was reported by researchers from Texas, U.S.A., raising concern about potential carcinogenic effects. In a similar investigation conducted in Wuerzburg, Germany, we did not find a comparable elevation of genomic damage. MPH is not genotoxic in standard test systems, but yielded one positive result in a rodent carcinogenicity study at the highest test dose only (60-fold above therapeutic doses). In conclusion, changes in treatment strategies do not seem justified currently. Larger studies are under way and will hopefully eliminate any remaining doubt about potential genotoxic or carcinogenic consequences of MPH treatment.

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Author information Authors and Affiliations
  1. Department of Toxicology, University of Wuerzburg, Wuerzburg, Germany

    H. Stopper

  2. Department of Child and Adolescent Psychiatry and Psychotherapy, University of Wuerzburg, Wuerzburg, Germany

    S. Walitza, A. Warnke & M. Gerlach

Authors
  1. H. Stopper
  2. S. Walitza
  3. A. Warnke
  4. M. Gerlach
Corresponding author

Correspondence to H. Stopper.

Additional information

Correspondence: Helga Stopper, Department of Toxicology, University of Wuerzburg, Versbacher Strasse 9, 97078 Wuerzburg, Germany

About this article Cite this article

Stopper, H., Walitza, S., Warnke, A. et al. Brief review of available evidence concerning the potential induction of genomic damage by methylphenidate. J Neural Transm 115, 331–334 (2008). https://doi.org/10.1007/s00702-007-0829-y

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