A RetroSearch Logo

Home - News ( United States | United Kingdom | Italy | Germany ) - Football scores

Search Query:

Showing content from https://link.springer.com/doi/10.1007/s002040050347 below:

Role of hydrazine in the mechanism of isoniazid hepatotoxicity in rabbits

Abstract

 Isoniazid (INH) continues to be a highly effective drug in the chemoprophylaxis and treatment of tuberculosis; however, its use is associated with hepatotoxicity (predominantly hepatic necrosis) in 1–2% of individuals. The INH metabolites, acetylhydrazine and hydrazine, have each been implicated as the causative hepatotoxin in INH-induced hepatotoxicity. Using a model of INH-induced hepatotoxicity in rabbits, in which INH-induced hepatotoxicity manifests as hepatic necrosis, hepatic steatosis (hepatic fat accumulation) and hypertriglyceridaemia (elevated plasma triglycerides), we compared the severity of these measures of toxicity with plasma levels of INH, acetylhydrazine and hydrazine. Plasma INH and acetylhydrazine were not correlated with markers of INH-induced hepatic necrosis or fatty changes. Plasma hydrazine at 32 h was correlated significantly with plasma argininosuccinic acid lyase (ASAL, a sensitive marker of hepatic necrosis) activity as area under the curve (r 2=0.54, P<0.002) and log plasma ASAL activity at 48 h after the first dose of INH (r 2=0.53, p<0.005), but not with fatty changes. These results show in this model of INH-induced hepatotoxicity in rabbits that hydrazine, and not INH or acetylhydrazine, is most likely involved in the pathogenic mechanism of hepatic necrosis.

This is a preview of subscription content, log in via an institution to check access.

Access this article Subscribe and save

Springer+ Basic

€34.99 /Month

Subscribe now Buy Now

Price includes VAT (Germany)

Instant access to the full article PDF.

Institutional subscriptions

Similar content being viewed by others Explore related subjectsDiscover the latest articles and news from researchers in related subjects, suggested using machine learning. Author information Authors and Affiliations
  1. Pharmacology and therapeutics, The University of British Columbia, Vancouver B.C., Columbia, , , , , , XX

    Troy C. Sarich, Mohammed Youssefi, Ting Zhou, Stephen P. Adams & Richard A. Wall

  2. Departments of Pharmacology and Therapeutics and Medicine, The University of British Columbia, 2176 Health Sciences Mall, Vancouver B.C., V6T 1Z3 Canada, , , , , , CA

    J. M. Wright

Authors
  1. Troy C. Sarich
  2. Mohammed Youssefi
  3. Ting Zhou
  4. Stephen P. Adams
  5. Richard A. Wall
  6. J. M. Wright
Additional information

Received: 21 February 1996/Accepted: 30 April 1996

About this article Cite this article

Sarich, T., Youssefi, M., Zhou, T. et al. Role of hydrazine in the mechanism of isoniazid hepatotoxicity in rabbits. Arch Toxicol 70, 835–840 (1996). https://doi.org/10.1007/s002040050347

Download citation


RetroSearch is an open source project built by @garambo | Open a GitHub Issue

Search and Browse the WWW like it's 1997 | Search results from DuckDuckGo

HTML: 3.2 | Encoding: UTF-8 | Version: 0.7.4