Bal-Price, A. and Brown, G. C., Inflammatory neurodegeneration mediated by nitric oxide from activated glia-inhibiting neuronal respiration, causing glutamate release and excitotoxicity.J. Neurosci., 21, 6480–6491 (2001).
Bell, M. J., Kochanek, P. M., Carcillo, J. A., Mi, Z., Schiding, J. K., Wisniewski, S. R., Clark, R. S., Dixon, C. E., Marion, D. W., and Jackson E., Interstitial adenosine, inosine, and hypoxanthine are increased after experimental traumatic brain injury in the rat.J. Neurotrauma. 15, 163–170 (1998).
Beltran, B., Mathur, A., Duchen, M. R., Erusalimsky, J. D., and Moncada, S., The effect of nitric oxide on cell respiration: A key to understanding its role in cell survival or death.Proc. Natl. Acad. Sci. U.S.A., 97, 14602–14607 (2000).
Choi, I. Y., Lee, S. J., Ju, C., Nam, W., Kim, H. C., Ko, K. H. and Kim, W. K., Protection by a manganese porphyrin of endogenous peroxynitrite-induced death of glial cellsvia inhibition of mitochondrial transmembrane potential decreaseGlia, 31, 155–164 (2000).
Delgado-Esteban, M., Almeida, A., and Bolanos, J. P., D-Glucose prevents glutathione oxidation and mitochondrial damage after glutamate receptor stimulation in rat cortical primary neurons.J. Neurochem., 75, 1618–1624 (2000).
Erecinska, M. and Silver, I. A., ATP and brain function.J. Cereb. Blood Flow Metab. 9, 2–19 (1989).
Gonzalez-Polo, R. A., Soler, G., Alonso, J. C., Rodriguez-Martin, A., and Fuentes, J. M., MPP+ causes inhibition of cellular energy supply in cerebellar granule cells.Neurotoxicology, 24, 219–225 (2003).
Gow, A. J., Thom, S. R., and Ischiropoulos, H., Nitric oxide and ONOO-mediated pulmonary cell death.Am. J. Physiol., 274, L112-L118 (1998).
Hagberg H., Andersson, P., Lacarewicz, J., Jacobson, I., Butcher, S. and Sandberg, M., Extracellular adenosine, inosine, hypoxanthine, and xanthine in relation to tissue nucleotides and purines in rat striatum during transient ischemia.J. Neurochem., 49, 227–231 (1987).
Haun, S. E., Segeleon, J. E., Trapp, V. L., Clotz, M. A., and Horrocks, L. A., Inosine mediates the protective effect of adenosine in rat astrocyte cultures subjected to combined glucose-oxygen deprivation.J. Neurochem., 67, 2051–2059 (1996).
Heales, S. J. and Bolanos, J. P., Impairment of brain mitochondrial function by reactive nitrogen species: the role of glutathione in dictating susceptibility.Neurochem. Int., 40, 469–474 (2002).
Ju, C., Yoon, K. N., Oh, Y. K., Kim, H. C., Shin, C. Y., Ryu, J. R., Ko, K. H., and Kim, W. K., Synergistic depletion of astrocytic glutathione by glucose deprivation and peroxynitrite: Correlation with mitochondrial dysfunction and subsequent cell death.J. Neurochem., 74, 1989–1998 (2000).
Jurkowitz, M. S., Litsky, M. L., Browning, M. J. and Hohl, C. M., Adenosine, inosine, and guanosine protect glial cells during glucose deprivation and mitochondrial inhibition: correlation between protection and ATP preservation.J. Neurochem., 71, 535–548 (1998).
Kobayashi, S., Conforti, L., Pun, R. Y. K. and Millhorn, D. E., Adenosine modulates hypoxia-induced responses in rat PC12 cellsvia A2A receptor.J. Physiol., 508, 95–107 (1998).
Litsky, M. L., Hohl, C. M., Lucas, J. H. and Jurkowitz, M. S., Inosine and guanosine preserve neuronal and glial cell viability in mouse spinal cord cultures during chemical hypoxia.Brain Res., 821, 426–432 (1999).
Lizasoain, I., Moro, M. A., Knowles, R. G., Darley-Usmar, V., and Moncada, S., Nitric oxide and ONOO− exert distinct effects on mitochondrial respiration which are differentially blocked by glutathione or glucose.Biochem. J., 314, 877–880 (1996).
Mazzio, E. and Soliman, K. F. A., D-(+)-glucose rescue against 1-methyl-4-phenylpyridinium toxicity through anaerobic glycolysis in neuroblastoma cells.Brain Res., 962, 48–60 (2003).
Michel, P. P., Marien, M., Ruberg, M., Colpaert, F., and Agid, Y., Adenosine prevents the death of mesencephalic dopaminergic neurons by a mechanism that involves astrocytes.J. Neurochem. 72, 2074–2082 (1999).
Packer, M. A. and Murphy, M. P., ONOO− formed by simultaneous nitric oxide and superoxide generation causes cyclosporin A-sensitive mitochondrial calcium efflux and depolarisation.Eur. J. Biochem., 234, 231–239 (1995).
Sims, N. R. and Anerson, M. F., Mitochondrial contributions to tissue damage in stroke.Neurochem. Int., 40, 511–526 (2002).
Shin, C. Y., Jang, E. S., Choi, J. W., Ryu, J. R., Kim, V. K., Kim, H. C., Choi, C. R., and Ko, K. H., Adenosine and purine nucleosides protect rat primary astrocytes from peroxynitritepotentiated, glucose deprivation-induced death: preservation of intracellular ATP level.Exp. Neurol., 176, 175–182 (2002).
RetroSearch is an open source project built by @garambo | Open a GitHub Issue
Search and Browse the WWW like it's 1997 | Search results from DuckDuckGo
HTML:
3.2
| Encoding:
UTF-8
| Version:
0.7.4