a A rat brain slice including needle tracts and regions of interest within the nucleus accumbens (NAc) in which immunoreactivity (IR) and cells were evaluated (1 core 1; 2 core 2; according to [29]). b BSI-B4-marked microglial cells after stab-wound injury (overview). c Quantification of the number of activated microglial cells after injury and microinjection of P2-receptor-agonists 2MeSATP, ADPβS, α,βmeATP (0.1 nmol each), and BzATP (0.3 nmol) after a postinjection time of 4 days. Values are expressed as percentage of ACSF-treated controls and represent mean ± SEM of six animals per group. d–f BSI-B4-IR of microglial cells in the NAc of rats, illustrating changes in the glial morphology: d A great number of resting (process-bearing) microglial cells (arrows) under control conditions; e, f changes in the number of resting microglial cells (arrow) and activated microglial cells (arrowhead) after different treatment conditions (scale bar = 200 μm). g Quantification of the effects of agonists 2MeSATP, ADPβS, α,βmeATP (0.1 nmol each), and BzATP (0.3 nmol) alone and in combination with PPADS (0.1 nmol/0.03 nmol each) or BBG (1 pmol) on BSI-B4-IR of microglial cells in brain slices of the NAc of the rat after a postinjection time of 4 days. Data are expressed as percentage of the ACSF-treated side and represent the mean ± SEM of six animals per group (*p < 0.05, vs. ACSF group; +p < 0.05, agonist vs. antagonist/agonist group)
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