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MGI - Inbred Strains: ICGN

Inbred Strains of Mice: ICGN

ICGN

Inbr. F26 (Asano, 1993). Albino. Origin: Outbred ICR stock inbred by Ogura,Takano, Yamamoto and Asano, Dept. of Veterinary Sci. NIH, Japan. All animals develop glomerular nephritis (

Ogura et al 1989

, 1990, 1991a,b). Initially, mice develop hyproteinemia and hypoalbuminemia with a progressive rise in urinary protein. Later the concentrations of total cholesterol, triglyceride and betalipoprotein increase. Finally there is a rapid deterioration of renal function with high blood urea nitrogen and creatinine levels (

Ogura et al, 1994

).

Ogura A., Asano T., Matuda J., Nakagawa M., and Fukui M. (1989) Characteristics of mutant mice (ICGN) with spontaneous renal lesions: a new model for human nephrotic syndrome. Lab. Anim. 23, 169-174.

Ogura A., Asano T., Suzuki O., Yamamoto Y., Noguchi Y., Kawaguchi H., and Yamaguchi Y. (1994) Hereditary nephrotic syndrome with progression to renal failure in a mouse model (ICGN strain): Clinical study. Nephron 68, 239-244.

INBRED STRAINS OF MICE
Updated 9 Apr. 1998
Michael FW Festing
MRC Toxicology Unit, Hodgkin Building,
University of Leicester, UK

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