Inbred Strains of Mice: AL
ALInbr: F166. Albino. Genet:
a, b, c.Believed to have originated from an illegitimate mating of strain A followed by b x s mating, but should not be considered as a substrain of strain A. Very low mammary tumour incidence (
Staats, 1976).
Normal physiology
High erythrocyte catalase level (3/18) (Hoffman and Rechcigle, 1971).
DrugsPhenobarbital i.p. does not induce hepatic epoxide hydrase (cf. 3/7) (
Oesch et al., 1973., 1973). Long hexobarbital sleeping time (9/9) and low liver hexobarbital oxidase level (1/9) (
Vesell, 1968).
ImmunologyResistant to the induction of liver amyloid, but a high level of spontaneous amyloidosis (10/10) (
Ram et al., 1969., 1969). Low susceptibility to induction of amyloid (6/6) (
Willerson et al., 1969., 1969). Good immune response to synthetic double-stranded RNA (1/7) (strain quoted as ALN, but presumed to be AL/N) (
Steinberg et al., 1971., 1971). Poor immune response to Vi antigen (cf. 3/5) (
Gaines et al., 1965., 1965). High anti-DNP antibody concentration (1/7) (
Paul et al., 1970., 1970).
ReproductionGood breeding performance with 2.3 young per female per month (5/24) (
Hansen et al., 1973., 1973).
MiscellaneousAL/N has only 38 chromosomes, including two translocation submetacentrics involving Robertsonian translocations of chromosomes 5 and 19 (
White and Tjio, 1975).
Gaines S., Currie J. A., and Tully J. G. (1965) Factors affecting formation of incomplete Vi antibody in mice. J. Bacteriol. 90, 635-642.Hoffman H. A. and Rechcigl M. Jr. (1971) Erythrocyte catalase in inbred mice. Enzyme 12, 219-225.
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